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Armour-GX5 (3.0) Batting Gloves (Red-Gold)
Armour-GX5 (3.0) Batting Gloves (Red-Gold) - Image 2

Armour-GX5 (3.0) Batting Gloves (Red-Gold)

(38 customer reviews)

₹1,599.00 – ₹1,649.00

The ARMOUR-GX5 (3.0) Cricket Batting Gloves is hybrid combination of Block & V-split style gloves, having 3 split finger for better flexibility. Overall covered with light weight quality PU with triangle & bar-split back design providing elegant and premium looks.

SKU: BG-GX5T-30-RDGL Categories: , Tag:
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Description

The ARMOUR-GX5 (3.0) Cricket Batting Gloves is hybrid combination of Block & V-split style gloves, having 3 split finger for better flexibility. Overall covered with light weight quality PU with triangle & bar-split back design providing elegant and premium looks.

Additional information

Color

GOLD, Red
Protection Range

GX (GLOVES)
Size

LARGE, MEDIUM, YOUTH
HAND / LEG

LEFT, RIGHT

38 reviews for Armour-GX5 (3.0) Batting Gloves (Red-Gold)

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    Restore a balanced microbiome – after infections, antibiotics or diet changes, kids can have an over‑growth of “bad” bacteria or a drop in beneficial species.
    A healthy gut supports digestion, immune function and even mood.

    Enhance barrier integrity – many strains help tighten the intestinal lining, reducing “leaky gut.”
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    Modulate immunity – certain probiotics can dampen over‑reactive immune
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    Immune Training Repeated exposure to microbial antigens educates innate immunity; this is
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    Confounding Factors – Diet, exercise, stress levels,
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    Causality vs Correlation – Observational data can only suggest associations; a study where participants are randomly assigned to “high‑exposure” vs “low‑exposure” provides stronger evidence that changes in exposure cause immune changes.

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    Recruitment & Screening

    – Advertise via community centers, universities,
    social media.
    – Screen for inclusion/exclusion criteria; obtain informed consent.

    Baseline Data Collection (Week 0)

    – Demographics, health status, lifestyle questionnaire.

    – Blood samples and stool samples (baseline).
    – Baseline questionnaires on stress, sleep, diet.

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    – Use computer-generated randomization lists; assign participants to intervention or control groups.

    – Provide instructions for the assigned activity (e.g., schedule for
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    – At weeks 2, 6, and 12: repeat blood and stool sampling, questionnaires.

    – Continuous monitoring of adherence via app logs or self-reports.

    Data Management

    – All data entered into secure electronic database (e.g., REDCap).

    – Regular backups; double-data entry for critical variables to
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    Statistical Analysis Plan

    – Primary analysis: mixed‑effects linear models comparing changes in metabolite levels between groups over time, adjusting for baseline values.

    – Secondary analyses: correlation of metabolite changes with microbiome composition shifts (16S rRNA data).

    – Multiple testing correction via Benjamini–Hochberg FDR.

    Reporting and Dissemination

    – Prepare manuscript following STROBE guidelines.

    – Present findings at relevant conferences; share data in public repositories (e.g.,
    MetaboLights).

    By rigorously applying this detailed protocol, researchers can confidently attribute observed metabolomic changes to specific dietary
    interventions, thereby enhancing reproducibility and advancing our understanding of diet–metabolism
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    *A Practical Guide for Primary Care Physicians*

    —

    ## 1. What Is Gynecomastia?

    | Feature | Typical Findings |
    |———|——————|
    | **Definition** | Hormonal imbalance between estrogen and androgen activity → proliferation of glandular breast tissue.
    |
    | **Epidemiology** | ~40 % lifetime prevalence in men; peaks at ages 15–20, 30–50 (secondary),
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    | **Clinical Picture** | Painless, firm or rubbery mass beneath the
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    Usually unilateral but can be bilateral. |
    | **Differential** | Fibroadenoma, cystic disease,
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    |

    —

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    | Condition | Key Features | Distinguishing Points
    vs Gynecomastia |
    |———–|————–|—————————————-|
    | **Benign fibroadenoma** | Solid mass, mobile, well‑circumscribed, often 2 cm, may fluctuate in size with
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    | **Phyllodes tumour** | Rapidly enlarging, can be >5–10 cm; may have a leaf‑like architecture on histology | Imaging:
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    | **Inflammatory breast cancer** | Sudden erythema, peau d’orange, rapid progression,
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    carcinoma cells in dermal lymphatics. |
    | **Metastatic disease** | Usually presents as multiple masses or
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    |———-|——————-|————————|————————–|
    | **Primary Breast Tumours** | • Invasive ductal carcinoma (IDC) – inflammatory variant
    • Inflammatory breast carcinoma (IBC)
    • Phyllodes tumour, especially malignant
    • Metaplastic carcinoma (fibroblastic, spindle cell)
    • Ductal carcinoma in situ (DCIS) with solid component |
    IDC is most common; IBC accounts for <2% but presents with erythema and rapid growth. Malignant phyllodes can mimic. Metaplastic carcinomas often present as firm masses and may show desmoplasia. DCIS may have calcifications but usually not such large mass. | IBC typically shows skin thickening, peau d'orange; no obvious calcifications. Phyllodes are biphasic with stromal proliferation; malignant variants can be large. Metaplastic carcinoma shows spindle cells and may express vimentin, EMA, cytokeratin. |
    | **Immunohistochemistry** | 1. **Pan‑keratin (AE1/AE3 or CK5/6)** – highlights epithelial component.
    2. **Cytokeratin 7 (CK7)** – usually positive in breast carcinoma.
    3. **Estrogen Receptor (ER), Progesterone Receptor (PR)** – to assess hormone status.
    4. **HER‑2/neu** – for targeted therapy.
    5. **Gross‑cystic disease fluid protein‑15 (GCDFP‑15) / Mammaglobin** – supportive of breast origin.
    6. **GATA3** – highly sensitive for mammary epithelium.
    7. **S100, HMB‑45** – to rule out melanocytic lesions if morphology ambiguous.

    The panel should be chosen based on the morphologic features: if classic ductal carcinoma is seen, ER/PR/HER‑2 are routine; if an unusual or borderline lesion appears, include GATA3 and GCDFP‑15.

    —

    ## 4. Special Considerations for Borderline/Uncertain Cases

    | Scenario | Morphologic Clues | Suggested IHC Panel | Interpretation |
    |———-|——————-|———————|—————-|
    | **Atypical ductal hyperplasia (ADH) vs DCIS** | Small clusters of cells with nuclear atypia but no comedonecrosis. | ER, PR, HER‑2, Ki‑67 | High ER/PR and low Ki‑67 support ADH; higher Ki‑67 suggests DCIS |
    | **Low‑grade papillary lesions** | Fibrin strands, small epithelial clusters. | S100 (to rule out melanocytic), TTF‑1 negative (exclude lung), CK5/6 | Positive for CK5/6 supports benign or low‑grade; high Ki‑67 indicates malignancy |
    | **Benign vs malignant phyllodes** | Similar stromal overgrowth, but malignant shows hypercellularity. | p53, CD34, Ki‑67 | Elevated p53 and Ki‑67 in malignant |

    —

    ## 5. Practical Workflow for a Pathology Lab

    1. **Specimen receipt & grossing**
    – Photograph, label, record size/weight.
    – Ensure representative sections.

    2. **Slide preparation**
    – Standard H&E.
    – Additional immunostains per panel above if morphology is ambiguous.

    3. **Microscopic evaluation**
    – Assess architecture, cytology, stroma, vascularity, margins.
    – Record findings in a standardized template (e.g., "benign epithelial proliferation with mild atypia; no invasion").

    4. **Immunohistochemistry decision tree**
    – If morphology suggests hyperplasia or adenoma → proceed with ER/PR panel.
    – If suspicious for carcinoma → add HER2, Ki-67, p63/cytokeratin 5/6, GATA3.

    5. **Reporting format**
    – Summary statement: e.g., "Benign endometrial hyperplasia without atypia; no evidence of malignancy."
    – Detailed description of histology and immunoprofile.
    – Recommendations (e.g., repeat sampling if clinical suspicion remains).

    —

    ### 4. Validation and Quality Assurance

    – **Pilot Run**: Process a batch of 10–20 routine samples, compare findings with standard pathology results to assess concordance.
    – **Inter‑operator Variability**: Have at least two technicians independently process the same sample; evaluate agreement on histological interpretation and immunoprofile.
    – **Control Samples**: Include known positive/negative control slides for each antibody run (e.g., a breast carcinoma tissue microarray).
    – **Documentation**: Maintain detailed SOP logs, calibration records for imaging equipment, and assay performance metrics.

    —

    ### 5. Clinical Implementation Timeline

    | Phase | Duration | Key Activities |
    |——-|———-|—————-|
    | Setup & Validation | 4–6 weeks | Procure reagents, validate protocols, train staff |
    | Pilot Testing | 2–3 weeks | Process real patient samples, compare with standard pathology |
    | Full Roll‑out | Ongoing | Integrate into routine diagnostic workflow, periodic quality checks |

    —

    ## Summary

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